What causes panic attacks and how can you prevent them Cindy J. Aaronson

The body becomes its own corset.

Past, present, and future
exist as a single force.

A swing without gravity soars
to a terrifying height.

The outlines of people and things
dissolve.

Countless poets and writers
have tried to put words

to the experience of a panic attack—

a sensation so overwhelming, many people
mistake it for a heart attack, stroke,

or other life-threatening crisis.

Though panic attacks don’t cause
long-term physical harm,

afterwards, the fear of another attack
can limit someone’s daily life—

and cause more panic attacks.

Studies suggest that almost a third of us

will experience at least one
panic attack in our lives.

And whether it’s your first,
your hundredth,

or you’re witnessing someone else
go through one,

no one wants to repeat the experience.

Even learning about them can
be uncomfortable, but it’s necessary—

because the first step to preventing
panic attacks is understanding them.

At its core, a panic attack
is an overreaction to the body’s

normal physiological response
to the perception of danger.

This response starts with the amygdala,

the brain region involved
in processing fear.

When the amygdala perceives danger,

it stimulates
the sympathetic nervous system,

which triggers the release of adrenaline.

Adrenaline prompts an increase
in the heart and breathing rate

to get blood and oxygen
to the muscles of the arms and legs.

This also sends oxygen to the brain,
making it more alert and responsive.

During a panic attack,

this response is exaggerated
well past what would be useful

in a dangerous situation,

causing a racing heart, heavy breathing,
or hyperventilation.

The changes to blood flow
cause lightheadedness

and numbness in the hands and feet.

A panic attack usually peaks
within 10 minutes.

Then, the prefrontal cortex
takes over from the amygdala

and stimulates
the parasympathetic nervous system.

This triggers the release of a hormone
called acetylcholine

that decreases the heart rate
and gradually winds down the panic attack.

In a panic attack, the body’s
perception of danger

is enough to trigger the response we would
have to a real threat— and then some.

We don’t know for sure
why this happens,

but sometimes cues in the environment
that remind us

of traumatic past experience
can trigger a panic attack.

Panic attacks can be part
of anxiety disorders

like PTSD, social anxiety disorder, OCD,
and generalized anxiety disorder.

Recurring panic attacks,
frequent worry about new attacks,

and behavioral changes
to avoid panic attacks

can lead to a diagnosis
of a panic disorder.

The two main treatments
for panic disorder

are antidepressant medication
and cognitive behavioral therapy, or CBT.

Both have about a 40% response rate—

though someone who responds to one
may not respond to the other.

However, antidepressant medications
carry some side effects,

and 50% of people relapse
when they stop taking them.

CBT, meanwhile, is more lasting,
with only a 20% relapse rate.

The goal of CBT treatment for panic
disorder is to help people learn

and practice concrete tools
to exert physical, and in turn mental,

control over the sensations and thoughts
associated with a panic attack.

CBT begins with an explanation of the
physiological causes of a panic attack,

followed by breath and muscle exercises
designed to help people

consciously control breathing patterns.

Next comes cognitive restructuring,

which involves identifying
and changing the thoughts

that are common during attacks—

such as believing you’ll stop breathing,
have a heart attack, or die—

and replacing them
with more accurate thoughts.

The next stage of treatment is exposure
to the bodily sensations and situations

that typically trigger a panic attack.

The goal is to change the belief,
through experience,

that these sensations and situations
are dangerous.

Even after CBT, taking these steps
isn’t easy in the grip of an attack.

But with practice, these tools
can both prevent and de-escalate attacks,

and ultimately reduce the hold of panic
on a person’s life.

Outside formal therapy,

many panickers find relief from the same
beliefs CBT aims to instill:

that fear can’t hurt you,
but holding on to it will escalate panic.

Even if you’ve never had a panic attack,

understanding them will help you identify
one in yourself or someone else—

and recognizing them is the first step
in preventing them.

身体变成了自己的紧身胸衣。

过去、现在和未来
作为一个单一的力量存在。

没有重力的秋千飙升
到一个可怕的高度。

人和事物的轮廓
消失了。

无数诗人和
作家试图用文字

来描述惊恐发作的经历——

这种感觉如此强烈,许多人
误以为是心脏病发作、中风

或其他危及生命的危机。

虽然惊恐发作不会造成
长期的身体伤害,但

之后,对另一次发作的恐惧
会限制某人的日常生活——

并导致更多的惊恐发作。

研究表明,几乎三分之一的人在我们的生活

中至少会经历一次
惊恐发作。

无论是你的第一次,
你的百分之一,

还是你正在目睹其他人
经历一次,

没有人愿意重复这种经历。

即使了解它们也会
让人不舒服,但这是必要的——

因为防止
恐慌发作的第一步是了解它们。

从本质上讲,惊恐发作
是对身体

对危险感知的正常生理反应的过度反应。

这种反应始于杏仁核,

即参与处理恐惧的大脑区域

当杏仁核感知到危险时,

它会
刺激交感神经系统,

从而触发肾上腺素的释放。

肾上腺素
促进心脏和呼吸频率的增加,

以将血液和氧气输送
到手臂和腿部的肌肉。

这也会向大脑输送氧气,
使其更加警觉和反应灵敏。

在惊恐发作期间,

这种反应被夸大了,
远远超过了

在危险情况下有用的反应,

导致心跳加速、呼吸沉重
或换气过度。

血流的变化

导致手脚头晕和麻木。

惊恐发作通常
在 10 分钟内达到高峰。

然后,前额叶皮层
接管杏仁核


刺激副交感神经系统。

这会触发一种叫做乙酰胆碱的激素的释放,这种激素

会降低心率
并逐渐缓解惊恐发作。

在惊恐发作中,身体
对危险

的感知足以触发
我们对真正威胁的反应——然后是一些反应。

我们不确定
为什么会发生这种情况,

但有时环境
中提醒我们

过去的创伤经历的线索
会引发恐慌症。

惊恐发作可能
是焦虑症的一部分,

如 PTSD、社交焦虑症、强迫症
和广泛性焦虑症。 反复

发作的惊恐发作、
经常担心新的发作

以及
避免惊恐发作的行为改变

可能导致
惊恐障碍的诊断。 恐慌症

的两种主要治疗
方法

是抗抑郁药物
和认知行为疗法,或 CBT。

两者都有大约 40% 的响应率——

尽管对其中一个做出响应的人
可能不会对另一个做出响应。

然而,抗抑郁药物
有一些副作用

,50% 的人
在停止服用后会复发。

与此同时,CBT 更持久
,只有 20% 的复发率。

CBT 治疗恐慌症的目标
是帮助人们学习

和练习具体的工具
来发挥身体,进而在精神上

控制
与恐慌发作相关的感觉和思想。

CBT 首先解释
惊恐发作的生理原因,

然后是
旨在帮助人们

有意识地控制呼吸模式的呼吸和肌肉锻炼。

接下来是认知重构,

这涉及识别
和改变发作期间常见的想法——

例如相信你会停止呼吸
、心脏病发作或死亡——


用更准确的想法代替它们。

下一阶段的治疗是接触

通常会引发惊恐发作的身体感觉和情况。

目标是通过经验改变人们对

这些感觉和情况
是危险的信念。

即使在 CBT 之后,采取这些步骤
也不是一件容易的事。

但通过实践,这些工具
既可以防止攻击,

也可以降低攻击,最终减少恐慌
对一个人的生活造成的影响。

在正式治疗之外,

许多恐慌者从 CBT 旨在灌输的相同信念中解脱出来

:恐惧不会伤害你,
但坚持下去会加剧恐慌。

即使您从未经历过惊恐发作,

了解它们也将帮助您识别
自己或他人中的一个——

而识别它们
是预防它们的第一步。