Can you outsmart the fallacy that fooled a generation of doctors Elizabeth Cox

Oh the humanity!

Ah… humanity…

It’s a trainwreck,
but I can’t look away.

It’s 1843, and a debate is raging
among physicians

about one of the most common
killers of women: childbed fever.

Childbed fever strikes
within days of giving birth,

killing more than 70% of those infected—
and nobody knows what causes it.

Obstetrician Charles Meigs has a theory.

Having observed abdominal inflammation

in patients who go on
to develop the fever,

he claims this inflammation
is the cause of childbed fever.

Much of the medical establishment
supports his theory.

Oh, come on!

They really leave me no choice
but to teach them some skepticism.

That’s better.

Now, Meigs, your argument is based
on a fallacy— the false cause fallacy.

Correlation does not imply causation:

When two phenomena regularly occur
together,

one does not necessarily
cause the other.

So you say women who have inflammation
also come down with childbed fever,

therefore the inflammation
caused the fever.

But that’s not necessarily true.

Yes, yes, the inflammation comes first,
then the fever,

so it seems like the inflammation
causes the fever.

But by that logic, since babies
usually grow hair before teeth,

hair growth must cause tooth growth.

And we all know that’s not true,
right?

Actually, don’t answer that.

A couple of different things
could be going on here.

First, it’s possible that fever
and inflammation are correlated

purely by coincidence.

Or, there could be a causal relationship
that’s the opposite of what you think—

the fever causes the inflammation,

rather than the inflammation
causing the fever.

Or both could share a common
underlying cause you haven’t thought of.

If I may, just what do you think
causes inflammation? Nothing?

It just is? Really?

Humor me for a moment in discussing
one of your colleague’s ideas—

Dr. Oliver Wendell Holmes.

I know, I know,
you don’t like his theory—

you already wrote
a scathing letter about it.

But let’s fill your students in, shall we?

Holmes noticed a pattern:
when a patient dies of childbed fever,

a doctor performs an autopsy.

If the doctor then treats a new patient,

that patient often comes
down with the fever.

Based on this correlation

between autopsies of fever victims
and new fever patients,

he proposes a possible cause.

Since there’s no evidence that the autopsy
causes the fever beyond this correlation,

he doesn’t jump to the conclusion
that autopsy causes fever.

Instead, he suggests that doctors
are infecting their patients

via an invisible contaminant
on their hands and surgical instruments.

This idea outrages most doctors,
who see themselves as infallible.

Like Meigs here, who refuses
to consider the possibility

that he’s playing a role
in his patients’ plight.

His flawed argument doesn’t leave any
path forward for further investigation—

but Holmes’ does.

It’s 1847, and physician Ignaz Semmelweis

has reduced the number
of childbed fever deaths

in a clinic from 12% to 1%
by requiring all medical personnel

to disinfect their hands after autopsies
and between patient examinations.

With this initiative, he has proven
the contagious nature of childbed fever.

Ha!

It’s 1879, and Louis Pasteur
has identified

the contaminant responsible
for many cases of childbed fever:

Hemolytic streptococcus bacteria.

Hmm, my fries are cold.

Must be because my ice cream melted.

哦人类!

啊……人类……

这是火车残骸,
但我无法移开视线。

现在是 1843 年,医生之间正在激烈

争论女性最常见的
杀手之一:产褥热。

产后热病
在分娩后几天内发作,

导致超过 70% 的感染者死亡——
没有人知道是什么原因造成的。

产科医生查尔斯·梅格斯有一个理论。

在观察到

继续
发烧的患者的腹部炎症后,

他声称这种炎症
是产褥热的原因。

许多医疗机构都
支持他的理论。

哦,来吧!

他们真的让我
别无选择,只能教他们一些怀疑。

这样更好。

现在,梅格斯,你的论点是
基于一个谬误——虚假原因谬误。

相关性并不意味着因果关系:

当两种现象经常同时发生
时,

一种不一定
会导致另一种。

所以你说有炎症的女性
也会产褥热,

所以炎症
引起了发烧。

但这不一定是真的。

是的,是的,首先是炎症,
然后是发烧,

所以看起来炎症
引起了发烧。

但是按照这个逻辑,由于婴儿
通常在长出牙齿之前长出头发,因此

头发的生长必然会导致牙齿的生长。

我们都知道那不是真的,
对吧?

实际上,不要回答这个问题。

这里可能会发生一些不同的事情

首先,发烧
和炎症可能

纯属巧合。

或者,可能存在与您认为的相反的因果关系——

发烧导致炎症,

而不是炎症
导致发烧。

或者两者都可能有一个
你没有想到的共同根本原因。

如果可以,您认为是什么
引起了炎症? 没有什么?

它只是? 真的吗?

讨论
一下你同事的一个想法——

奥利弗·温德尔·霍姆斯博士,让我幽默一下。

我知道,我知道,
你不喜欢他的理论——

你已经写
了一封严厉的信。

但是,让我们填补你的学生,好吗?

福尔摩斯注意到一个规律:
当病人死于产褥热时

,医生会进行尸检。

如果医生随后治疗新患者,则

该患者通常
会发烧。

基于

发烧受害者
和新发烧患者尸检之间的这种相关性,

他提出了一个可能的原因。

由于没有证据表明尸检
导致发烧超出了这种相关性,因此

他没有草率地
得出尸检会导致发烧的结论。

相反,他建议医生

通过
手和手术器械上的无形污染物感染患者。

这个想法激怒了大多数医生,
他们认为自己是万无一失的。

就像这里的梅格斯一样,他
拒绝考虑

他在患者困境中扮演角色的可能性。

他有缺陷的论点没有
为进一步调查留下任何前进的道路——

但福尔摩斯的论点却有。

1847 年,医生 Ignaz Semmelweis

通过要求所有医务人员在尸检后和患者检查之间对双手进行消毒,将诊所
的产褥热死亡

人数从 12% 减少到 1%

通过这一举措,他已经证明
了产褥热的传染性。

哈!

那是 1879 年,路易斯·巴斯德(Louis Pasteur
)确定


导致许多产褥热病例的污染物:

溶血性链球菌。

嗯,我的薯条很冷。

一定是因为我的冰淇淋融化了。