What is obesity Mia Nacamulli

The most basic function of bodily fat
is self-storage of food reserves.

In prehistoric times, natural selection
favored genotypes

that could endure harsh conditions
by stocking the most fat.

With chronic malnutrition being
the norm for most of human history,

genetics evolved to favor fat storage.

So when did body fat become problematic?

The negative impacts of being overweight
were not even noted in medical literature

until as late as the 18th century.

Then, technological advances coupled
with public health measures

resulted in the betterment of the
quantity, quality, and variety of food.

Sustained abundance of good food
enabled a healthier population

to boom economically.

Output increased,
and with it, leisure time

and waistlines.

By the mid 19th century, being excessively
overweight, or obese,

was recognized as a cause of ill health,

and another century later,
declared deadly.

What is the distinction between being
overweight and being obese?

A calculation called the BMI
breaks it down for us.

For example, if someone weighs
65 kilgorams

and is 1.5 meters tall,

they have a BMI of about 29.

Obesity is a condition of excess body fat

that occurs when a person’s BMI
is above 30,

just over the overweight range
of 25 to 29.9.

While BMI can be a helpful estimate
of healthy weight,

actual body fat percentage can only
really be determined

by also considering information
like waist circumference

and muscle mass.

Athletes, for instance, have a naturally
higher BMI.

So how does a person become obese?

At its most basic, obesity is caused
by energy imbalance.

If the energy input from calories

is greater than the energy output
from physical activity,

the body stores the extra calories as fat.

In most cases, this imbalance comes
from a combination of circumstances

and choices.

Adults should be getting at least
2.5 hours of exercise each week,

and children a whole hour per day.

But globally, one in four adults
and eight out of ten adolescents

aren’t active enough.

Calorie-dense processed foods
and growing portion sizes

coupled with pervasive marketing

lead to passive overeating.

And scarce resources,

and a lack of access to healthy,
affordable foods

creates an even greater risk
in disadvantaged communities.

Yet, our genetic makeup also plays a part.

Studies on families and on separated twins

have shown a clear causal hereditary
relationship to weight gain.

Recent studies have also found
a link between obesity

and variations in the bacteria species
that live in our digestive systems.

No matter the cause, obesity is
an escalating global epidemic.

It substantially raises the probability
of diseases,

like diabetes,

heart disease,

stroke,

high blood pressure,

and cancer.

It affects virtually all ages, genders,
and socioeconomic groups

in both developed
and developing countries.

With a 60% rise in child obesity globally
over just two decades,

the problem is too significant to ignore.

Once a person is obese, the climb
to recovery becomes progressively steeper.

Hormonal and metabolic changes reduce
the body’s response to overeating.

After losing weight, a formerly overweight
person burns less calories

doing the same exercises

as a person who is naturally
the same weight,

making it much more difficult
to shed the excess fat.

And as people gain weight,

damage to signaling pathways makes it
increasingly difficult

for the brain to measure food intake
and fat storage.

There is, however, some evidence

that well-monitored,
long-term changes in behavior

can lead to improvements
in obesity-related health issues.

And weight loss from sustained
lifestyle changes,

or invasive treatments
like bariatric surgery,

can improve insulin resistance
and decrease inflammation.

What was once an advantage for survival
is now working against us.

As the world’s population continues
to slow down and get bigger,

moving and consciously eating our way
towards a healthier weight

is essential to our overall well-being.

And with the epidemic affecting
every country in the world

for different socioeconomic reasons,

obesity cannot be seen
as an isolated issue.

More global measures for prevention

are essential to manage
the weight of the world.

身体脂肪最基本的功能
是自我储存食物储备。

在史前时代,自然选择
偏爱

能够
通过放养最多脂肪来承受恶劣条件的基因型。

由于慢性营养不良
是人类历史上大部分时间的常态,

遗传学演变为有利于脂肪储存。

那么体脂是什么时候出现问题的呢? 直到 18 世纪

,医学文献才注意到超重的负面影响

然后,技术进步
加上公共卫生措施

导致
食物的数量、质量和种类得到改善。

持续丰富的优质食物
使更健康的人口

在经济上蓬勃发展。

产量增加了
,随之

而来的是休闲时间和腰围。

到 19 世纪中叶,过度
超重或肥胖

被认为是导致健康不佳的原因,

而另一个世纪后,又被
宣布为致命的。

超重和肥胖有什么区别?

一个称为 BMI 的计算
为我们分解了它。

例如,如果某人体重
65 公斤

,身高 1.5 米,

那么他们的 BMI 约为 29。

肥胖是一种身体脂肪过多

的情况,当一个人的
BMI 高于 30,

刚好超过
25 到 29.9 的超重范围时,就会出现这种情况。

虽然 BMI 可以帮助
估计健康体重,但

实际体脂百分比只能

通过同时考虑
腰围

和肌肉质量等信息来确定。

例如,运动员的
体重指数自然较高。

那么一个人是怎么变胖的呢?

从根本上说,肥胖是
由能量不平衡引起的。

如果卡路里输入的能量

大于身体活动的能量输出

,身体会将多余的卡路里储存为脂肪。

在大多数情况下,这种不平衡
来自环境

和选择的结合。

成人每周至少应进行
2.5 小时的运动

,儿童每天应进行整整一小时的运动。

但在全球范围内,四分之一的成年人
和十分之八的

青少年不够活跃。

卡路里密集的加工食品
和不断增加的份量

加上普遍的营销

导致被动暴饮暴食。

稀缺的资源,

以及无法获得健康、
负担得起的食物

,给弱势社区带来了更大的风险。

然而,我们的基因构成也发挥了作用。

对家庭和分居双胞胎的

研究表明,体重增加与遗传有明显的因果
关系。

最近的研究还发现
肥胖

与生活在我们消化系统中的细菌种类的变异之间存在联系

不管是什么原因,肥胖都是
一种不断升级的全球流行病。

它大大增加了

患糖尿病、

心脏病、

中风、

高血压

和癌症等疾病的可能性。

它几乎影响发达国家和发展中国家的所有年龄、性别
和社会经济群体

在短短二十年内,全球儿童肥胖率增加了 60%

这个问题太严重了,不容忽视。

一旦一个人肥胖,
恢复的过程就会变得越来越陡峭。

荷尔蒙和新陈代谢的变化会
降低身体对暴饮暴食的反应。

减肥后,以前超重的

在做与自然体重相同的人相同的运动时燃烧的卡路里更少

,因此
更难减掉多余的脂肪。

随着人们体重增加,

信号通路受损

使大脑越来越难以测量食物摄入量
和脂肪储存量。

然而,有一些证据

表明,受到良好监控
的长期行为变化

可以改善
与肥胖相关的健康问题。

通过持续的
生活方式改变

或减肥手术等侵入性治疗来减轻体重

可以改善胰岛素抵抗
并减少炎症。

曾经是生存优势的东西
现在对我们不利。

随着世界人口
继续放缓并变得更大,

移动并有意识地饮食以
实现更健康的

体重对于我们的整体福祉至关重要。

由于流行病因不同的社会经济原因影响
到世界上每个国家

肥胖不能被
视为一个孤立的问题。

更多的全球预防

措施对于管理
世界的重量至关重要。