What is dyslexia Kelli SandmanHurley

Take a moment to read the following.

How was that?

Frustrating?

Slow?

What were those sentences about?

They’re actually a simulation

of the experience of dyslexia,

designed to make you decode each word.

Those with dyslexia experience
that laborious pace

every time they read.

When most people think of dyslexia,

they think of seeing letters
and words backwards,

like seeing “b” as “d” and vice versa,

or they might think people with dyslexia

see “saw” as “was”.

The truth is people with dyslexia

see things the same way as everyone else.

Dyslexia is caused by a phonological
processing problem,

meaning people affected by it

have trouble not with seeing language

but with manipulating it.

For example, if you heard the word cat

and then someone asked
you, “Remove the ‘c’,”

what word would you have left?

At.

This can be difficult
for those with dyslexia.

Given a word in isolation,

like fantastic,

students with dyslexia
need to break the word

into parts to read it:

fan,

tas,

tic.

Time spent decoding makes it hard

to keep up with peers

and gain sufficient comprehension.

Spelling words phonetically,

like s-t-i-k

for stick

and f-r-e-n-s

for friends

is also common.

These difficulties are more
widespread and varied

than commonly imagined.

Dyslexia affects up to one in five people.

It occurs on a continuum.

One person might have mild dyslexia

while the next person has
a profound case of it.

Dyslexia also runs in families.

It’s common to see one family member

who has trouble spelling

while another family member

has severe difficulty decoding
even one syllable words,

like catch.

The continuum and distribution of dyslexia

suggests a broader
principle to bear in mind

as we look at how the brains
of those with dyslexia

process language.

Neurodiversity is the idea

that because all our brains
show differences

in structure and function,

we shouldn’t be so quick to label

every deviation from “the norm”

as a pathological disorder

or dismiss people living
with these variations

as “defective.”

People with neurobiological
variations like dyslexia,

including such creative
and inventive individuals

as Picasso,

Muhammad Ali,

Whoopi Goldberg,

Steven Spielberg,

and Cher,

clearly have every capacity

to be brilliant and successful in life.

So, here’s the special way

the brains of those with dyslexia work.

The brain is divided into two hemispheres.

The left hemisphere is generally
in charge of language

and, ultimately, reading,

while the right typically
handles spatial activities.

fMRI studies have found

that the brains of those with dyslexia

rely more on the right
hemisphere and frontal lobe

than the brains of those without it.

This means, when they read a word,

it takes a longer trip through their brain

and can get delayed in the frontal lobe.

Because of this neurobiological glitch,

they read with more difficulty.

But those with dyslexia

can physically change their brain

and improve their reading

with an intensive,
multi-sensory intervention

that breaks the language down

and teaches the reader to decode

based on syllable types
and spelling rules.

The brains of those with dyslexia

begin using the left hemisphere

more efficiently while reading,

and their reading improves.

The intervention works

because it locates dyslexia appropriately

as a functional variation in the brain,

which, naturally, shows
all sorts of variations

from one person to another.

Neurodiversity emphasizes this spectrum

of brain function in all humans

and suggests that to better
understand the perspectives

of those around us,

we should try not only to see
the world through their eyes

but understand it through their brains.

花点时间阅读以下内容。

怎么样?

令人沮丧?

慢的?

那些句子是关于什么的?

它们实际上是

对阅读障碍体验的模拟,

旨在让您解码每个单词。

那些有阅读障碍的人每次阅读时都会经历
那种费力的步伐

当大多数人想到阅读障碍时,

他们会想到倒着看字母
和单词,

例如将“b”视为“d”,反之亦然,

或者他们可能认为患有阅读障碍的人

将“saw”视为“was”。

事实上,患有阅读障碍的人

看待事物的方式和其他人一样。

阅读障碍是由语音
处理问题引起的,

这意味着受其影响的

人不仅无法看到语言,

而且无法操纵它。

例如,如果你听到 cat 这个词

,然后有人问
你,“去掉‘c’,”

你会留下什么词?

在。

对于有阅读障碍的人来说,这可能很困难。

给定一个孤立的词,

比如fantasy,

有阅读障碍的学生
需要将这个词

分成几部分来阅读:

fan、

tas、

tic。

解码所花费的时间使得

难以跟上同行

并获得足够的理解。

拼音拼写单词,

如 s-t-i-k

表示棍子

,f-r-e-n-s

表示朋友

也很常见。

这些困难比通常想象的更为
广泛和多样

阅读障碍影响多达五分之一的人。

它发生在一个连续统一体上。

一个人可能有轻度阅读障碍,

而另一个人则
有严重的阅读障碍。

阅读障碍也存在于家庭中。

通常会看到一个家庭

成员拼写有困难,

而另一个家庭

成员甚至很难解码
一个音节单词,

比如 catch。

阅读障碍的连续性和分布

表明了一个更广泛的
原则,

当我们研究
阅读障碍者的大脑如何

处理语言时,需要牢记这一点。

神经多样性的想法

是,由于我们所有的大脑

在结构和功能上都表现出差异,

我们不应该这么快将

每一次偏离“规范”的行为都标记

为病理性疾病,

或者将
患有这些变异的人

视为“有缺陷的”。

患有
阅读障碍等神经生物学变异的人,

包括毕加索、

穆罕默德·阿里、

乌比·戈德堡、

史蒂文·斯皮尔伯格

和雪儿等富有创造力和创造力的人,

显然有能力

在生活中取得辉煌和成功。

所以,这是

阅读障碍者大脑工作的特殊方式。

大脑分为两个半球。

左半球
通常负责语言

,最终负责阅读,

而右半球通常
负责空间活动。

功能磁共振成像研究发现

,与没有阅读障碍的人相比,患有阅读障碍的人的大脑

更多地依赖于右
半球和额叶

这意味着,当他们阅读一个单词时,

他们的大脑需要更长的时间,

并且可能会在额叶延迟。

由于这种神经生物学故障,

他们阅读起来更加困难。

但是那些有阅读障碍的人

可以通过密集的多感官干预来改变他们的大脑

并提高他们的阅读能力

,这种
干预

可以分解语言

并教读者

根据音节类型
和拼写规则进行解码。

阅读障碍患者的大脑在阅读时

开始

更有效地使用左半球

,他们的阅读能力也有所提高。

干预之所以奏效,

是因为它将阅读障碍恰当地定位

为大脑中的一种功能变异

,自然而然地,它显示

了人与人之间的各种变异。

神经多样性强调

所有人类的大脑功能谱,

并建议为了更好地
理解

我们周围人的观点,

我们不仅应该尝试
通过他们的眼睛看世界,

还应该通过他们的大脑来理解它。